There are two benefits from using ancillaries that either prevent estrogen formation from aromatiable AAS like test or dbol (arimidex) or block the estrogen from exerting its effect in some tissues (clomid) - 1) gyno prevention; 2) maintaining some degree of testicular function during the cycle, which prevents testicular atrophy (if the cycle is kept to a reasonable length, like 8-12 weeks) and thus makes it easier to restore the body'w own test production post-cycle and to keep your gains. Even if you knew for a fact that you don't get gyno easily, which you don't know, it's still a good idea to use one of these two. If you use clomid, then just continue it at 50 mg a day post-cycle for a month - you won't have to take a bigger dose the first day of your PCT because you'll already have it at the needed level in your system.

You don't have to do everything perfectly, and you won't in your first cycle. Just do the main things right - an effective dose of two synergistic AAS and, if they aromatise, which both do in this case, use some estro control. And a simple PCT. You could turn it into rocket science, but there's no need at our level.

Taking anti-estros throughout the cycle won't affect gains but will help keep them when the cycle is over.

Dbol stacks very well with test, they work synergistically when combined - the effect is higher than the sum of the effects of each drug if taken on their own. This is the basis under stacking two or more drugs - you don't simply pile some different compounds randomly together just to get a bigger total dosage. You choose drugs that comlement each other. One must do what the other dosen't do very well. There are, with some degree of simplification, 2 classes of AAS. Class I drugs bind well to the androgen receptors that are present in every cell in the body, and that's how they exert their anabolic effects (e.g. tren, nandrolone, anavar). Class II drugs bind weaky to the AR or not at all and so must have other ways through which they work, most of which aren't yet clearly established (e.g. dbol, anadrol). Test is the only AAS, or one of the very few, that acts significantly via both mechanisms. For synergistic effect, you combine a class I and a class II drug (test can take the place of the class I drug to produce a very good stack because test binds very well to the AR). That's why test stacks so well with dbol - the addition of dbol (or anadrol) to even an already high dose of test still results in a lot of added effect. Or why deca (nandrolone) plus dbol is such a classic stack. And that's why stacking two drugs from the same class achieves nothing other than a bigger total dosage, generally. And that's why test combines so well with pretty much everything - because it covers both AR-mediated and non-AR-mediated mechanisms. It's also why test works so well when taken alone.

I can't say anything about the quality of Veyron's test - I have only used pharma-grade test.

I think GH production doesn't halt until we're dead But I'm sure you know that.

I've never heard about dbol damaging GH production in users younger than 25. But I'm not very knowledgeable about this. Could you elaborate? I'd appreciate that.