Sorry to hear about your kid Nov.
Had that a while back too.
We named the kiddo, lit a sky lantern, said goodbye. It helps.
Best wishes to you and herself.

You can be tired of the implications all you want, but you are incorrect and you are actually the one spouting total garbage. Here is why:

1. Different compounds cause different durations of shut down. This is analogous to a more severe shut down at 1 end of the spectrum compared to the other
2. The longer the duration of a shut down the more deflated testicular cells can become. This in turn may cause a need for a more aggressive reboot and is therefore a harder shutdown
3. The longer the system is shut down the greater the chance of testicular cell death (lower likelihood than than shrinking excessively, but it does happen). This makes full return to baseline impossible and is therefore a harder shutdown
4. The longer the duration a system is exposed to excessive exogenous hormones, the more likely other parts of the cascade become affected and shut down. This is a wider shutdown and in the case of things like prolactin, they rebound slower than estrogen, which is turn rebounds slower than testo, therefore the system reboot is overall under a harder shut down.
5. In the cases of those naturally predisposed to lowering levels of endogenous testo over time, each exposure to exogenous hormones can lead to a more limited recovery. With each cycle recovery is more difficult and therefore the shutdown is harder
6. As a thought experiment, look twice at the usage of hcg. It is used to make testicular shutdown less severe. This naturally removes the notion of being shutdown or not, because the testes are working but other parts of the axis are not (the system is more complicated than shutdown or not, black and white etc.)
7. All humans are not all the same, have different metabolisms, some have disease some don't, some are at different stages of life than others
8. Time makes fools of us all in the end

We could keep going. This stuff is all really well documented, whether it is in the endo literature, the AAS studies, the TRT literature, the anecdotes on forums like this, peak testosterone etc, and also in the bloods posted in places like eroids.

I would also agree. If the faucet is turned off it is just off. Some drugs stay longer in your system like deca and EQ. So the wait time is a little longer to start pct. Usually why people stop said drugs a few weeks before the testosterone. But the faucet is still just shut. Maybe a 9 year old girl shut it or some huge meat head shut it. Difference is it can be opened back up one scenario will be a little more struggle to get the valve to turn. But off is off not one off is way more off.

I agree. Throw sarms in there to.
If your shut down, your shut down.

I find these topics amusing. Imo if your concerned about fertility or hair loss, don’t use steroids.. before you know it your taking as many drugs to combat the sides as you are for gains.

My hair was gone way before steroids. Started steroids at 42