posted Thu, 04/02/2015 - 02:02
2664
CJC-1295 w/ DAC
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Whats the minimal dose i can get away with using and still see results with cjc w/ dac? For the price of it i was hoping i could get away with 1mg per week. And about how long does it take to start seeing results? Im hoping its not mo ths like gh, i know most other peps kick in a little faster than that. Also is cjc w/ dac worth it and an effective pep alone or stacked?
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Also can email me at [email protected] tyvm
Hello to all I am very new to all of this so please cut me some slack, I was advised by some one I met at a doctors office to check out these peps for a speedy recovery and im clueless on all, please help[ in anyway u can........on 4/1 i wrecked a go cart and fractured my radius ulna and have some minor ligament damage and still waiting on decisions of surgery while road rash heals. I already ordered cjc-1295 dac 2mg and GHRP-2 5mg, considering ordering tb-500,bpc 157, ipamorelin also. I have no clue on any of the doses and administration whether it would be SQ, IM, IV and if they need to be administered separately or if they can be combined in to one solution. I am a FF/medic and am comfortable with administering any route and good with the formulas just need a lil help. Please i need to get back to work ASAP so any advise will help. TYVM in advance
pepmaneuropeI am currently runnning this at 4mg per week. At week 2 I noticed my weight started to shoot up. My pumps in the gym and my sleep has dramatically improved.
I look much fuller and leaner now. I'll be running the 4mg per week for 8 weeks.
Stack a ghrp (my favorite is ghrp2) alongside your cjc-1295 add in huperzine-A and egcg every time your pin ghrp (3x / day) and you will have constant high dose pulsing of hgh all day long. I've done this exact stack with excellent results. I start to notice better sleep with the first few weeks. Your body starts to take on a "different" look. Very subtle, your muscles will feel full but right, skin will look better, look tighter, look leaner. Your face will flush every time you pin good Cjc so look out for that. I usually start the see the bulk of the results around week 3-4. Scale will go up mostly from water retention but you will look leaner in the mirror. I was running cjc 2mg/week ghrp2 150mcg 3x/ day. I think you could get away with 1mg /week but with cjc the more the better. I've seen studies done with 250mcg/kg of body weight of cjc-1295 and serum levels of hgh were that of 15-20iu of PHARMA grade gh.
Have a read you might find this interesting
Default Huperzine A in conjunction with GHRP & GHRH use.
This was briefly mentioned in the quick answer/question dump, but I believe there may exist the possibility that Hup A could interfere with the use of GHRP and GHRH. Hup A is a fairly potent acetylcholinesterase inhibitor (1). Acetylcholinesteraze (AChE) is an enzyme that degrades acetylcholine (2). Acetylcholine suppresses the release of endogenous somatostatin (3).
Subsequent to the above, Hup A is touted as a GH stimulant due to the suppression of somatostatin. The half life of Hup A is 4-6 hours and GH pulses are on average 3 hours apart. My concern is that if we suppress the release of somatostatin we are possibly interfering with the axis. The pituitary needs time to refill it's store of GH for the next pulse. I am curious if this suppression of somatostatin creates an environment where we have a static level of GH leaking from the pituitary rather than a pulse.
There were studies done that show that the use of an acetylcholinesterase actually increased the volume of the GH pulse:
http://www.life-enhancement.com/maga...gsnew-benefits
Two new studies1,2 by two independent groups using donepezil reveal exciting new effects in humans. Donepezil was chosen for the first study1 because it is the most widely used selective cerebral cholinesterase inhibitor for Alzheimer’s, with a low rate of side effects. The first study1 was a randomized, controlled study of donepezil in 24 healthy elderly males 61–70 years of age. The effects of donepezil at 5 mg/day for 4 weeks and then 10 mg/day for another 4 weeks were determined for GH release and IGF-1 levels.The authors cite an earlier study in which the cerebral-selective cholinesterase inhibitor rivastigmine powerfully enhanced GH release in response to repeated GHRH (growth hormone-releasing hormone) pulses in healthy elderly male and female subjects. The mechanism for this potentiation is probably via the inhibition of somatostatin, a hormone that inhibits growth hormone release from the pituitary.1 One benefit of using a selective cholinesterase inhibitor to increase GH release as compared to using GH itself is (aside from the cost and inconvenience of injections) that the GH release in response to the cholinesterase inhibitor is still subject to natural regulatory mechanisms, so that the GH is released when it normally would be, only more so, whereas GH treatments cause GH effects without control mechanisms in place to limit it to physiological functionality. As a consequence, human treatment with actual GH has been fraught with side effects, such as edema, arthralgia, myalgia, and nerve-compression syndromes,1 as well as increased blood sugar levels (reduced glucose tolerance).
Original study here:
http://www.sciencedirect.com/science...31556504003353
References and links:
(1) Huperzine A, a novel promising acetylcholinesterase inhibitor.
(2) Acetylcholinesterase
(3) Effects of direct and indirect acetylcholine receptor agonists on growth hormone secretion in humans
interesting. very good read bro. im going to have to look into this more