This is the missing piece now we can see what actually moved instead of guessing, and the comparison tells a much clearer story than the mid-cycle numbers alone did. One caveat your baseline was drawn end of week 1, so you already had test and dbol on board, meaning it's not a true pre-cycle baseline your real natural starting point was likely even cleaner. But it's close enough to week 1 to be a useful reference. Here's what the comparison actually shows.
Your liver: the enzyme rise is real but it's the expected dbol/accutane story, and it's not alarming in context.
Week 1: ALT 26, AST 21 (both normal). Week 10: ALT 85, AST 52. So your liver enzymes roughly tripled over the cycle. That sounds dramatic, but it's the textbook response to running dbol (escalating to 40mg) plus the accutane you added at week 3 both hit the liver, and a tripling into the 80s for ALT on an oral plus accutane is within the range you'd expect, not a red flag for liver failure. It's elevated, it's worth bringing down, but it's a predictable drug effect, not a sign something's gone wrong. And critically: you started from genuinely healthy liver values (26/21), so you had room, and now that the dbol's done and you've dropped the accutane, these should fall back toward your week-1 baseline. The week 1 numbers prove your liver was fine before the orals so this is reversible drug effect, not underlying damage. Scale up the TUDCA to 1000mg and add NAC 1200mg to help it along, and recheck if it's not trending back down off the dbol and accutane, then investigate further, but expect recovery.
Your lipids: this is the real story, and the baseline shows exactly how much the dbol moved them.
Week 1: HDL 39, non-HDL 138, ratio 4.5, total cholesterol 177. Week 10: HDL 27, non-HDL 177, ratio 7.6, total 204. So the dbol dropped your HDL from 39 to 27 and pushed your ratio from a healthy 4.5 to a high 7.6. That's a significant lipid shift, and the baseline confirms it's the cycle (specifically the dbol) driving it, not where you started you started in a reasonable place and the oral moved you into high-risk territory. This is the marker that took the biggest hit. The reassuring part: your week-1 HDL of 39 and ratio of 4.5 show your lipids recover to a decent baseline off the orals, so once the dbol fully clears, HDL should climb back toward that 39. Scale up the support fish oil to 4g, citrus bergamot 1200mg and the week 10 recheck (off the dbol) should show recovery. If HDL hasn't moved back up meaningfully, that's the low-dose statin conversation, but give the dbol clearance time to work first.
IGF-1: here's something genuinely useful the baseline reveals.
Week 1: IGF-1 189. That's your reference. When your pending IGF-1 from the current bloods comes back, you can compare against 189 to see what your GH/growth status is doing on cycle. Without the baseline you'd have had no idea if a number was high or low for you now you do. Worth noting 189 at week 1 is a solid IGF-1 level.
E2: the baseline gives you your starting point, and it matters with your low SHBG.
Week 1: E2 12 (on a standard assay, and you had test on board already). That's actually on the lower side for week 1, which is interesting. When your pending E2 comes back, compare to this 12 but remember with deca and dbol in the mix and your SHBG crashed, interpreting E2 is trickier (the 19-nors affect the picture, and standard E2 assays can be thrown off). The key point from the baseline: your E2 wasn't running high at week 1, so if it's climbed significantly by week 10, that's the cycle pushing it. Get the pending E2 and compare.
Prolactin: the baseline makes the deca-watch concrete.
Week 1: prolactin 3.4. Week 10: prolactin 13. So prolactin nearly quadrupled over the cycle. It's still technically in range at 13, but the baseline shows it's clearly climbing on the deca it went from 3.4 to 13, and you've got 8 more weeks of deca to go. This is exactly the trend to stay ahead of. The jump from 3.4 to 13 is the deca's progestogenic activity expressing itself, and on an 18-week run it may keep climbing. This is your concrete reason to get ahead of it now with the P5P/L-tyrosine and to seriously consider the masteron the baseline confirms prolactin is on the move, not stable.
SHBG: confirms the expected suppression.
Week 1: SHBG 20. Week 10: SHBG 10. Halved over the cycle, expected from the androgen load. This matters for reading your free T and E2 (more free hormone), as I told you and the baseline shows it dropping, so your free hormone fraction is rising through the cycle.
The genuinely reassuring stuff the baseline confirms:
HbA1c: 4.6 → 5.0, both excellent, no glucose problem developing. Hematocrit: 49.1 → 46.4 (actually went slightly down, no polycythemia, good though it can climb later). Hemoglobin 16.1 → 14.9, fine. Platelets 321 → 399, both normal. Thyroid stable and in range across both. Free test was 304 with total 1281 at week 1 (high free T, expected on cycle with low SHBG). DHEA 375 at baseline, normal. So your glucose, blood counts, thyroid, and kidney picture are all holding up well the baseline confirms the only things that meaningfully moved in a concerning direction are lipids (the big one), liver enzymes (expected, reversible), and prolactin (climbing on deca, watch it).
What the baseline changes about your plan:
Mostly it's reassuring it shows you started healthy and the changes are identifiable drug effects, not underlying problems, which means they should reverse. The three things to act on: lipids (scale up support, expect HDL recovery off the dbol, statin conversation only if it doesn't recover), liver (TUDCA up to 1000mg, add NAC, expect recovery now the dbol/accutane are done), and prolactin (it went 3.4→13, it's climbing on the deca, get ahead of it with P5P/L-tyrosine and consider masteron, watch for gyno signs). Everything else glucose, blood counts, thyroid the baseline confirms is fine.
So the headline: the baseline shows you started from a healthy place, and the cycle moved your lipids hard (HDL 39→27), your liver enzymes predictably (tripled but reversible), and your prolactin notably (3.4→13, climbing on deca). None of it is alarming given you started healthy and the main drivers (dbol, accutane) are now done but the lipids need support and time, the liver needs the upgraded support, and the prolactin is the trend to stay ahead of on your remaining 8 weeks of deca. Get the pending E2 and IGF-1 and compare to your week-1 numbers (12 and 189) to complete the picture. Good that you got this baseline it turns guesswork into an actual before-and-after.

Smart call dropping it, and the timing's right dbol's done so the main acne driver is winding down anyway, and you'd already had reservations about the accutane, so trusting that instinct was the right move. The back acne flaring was the dbol-driven androgenic surge plus the dose escalation, that should settle now the oral's out. Here's how to manage it without the accutane, because you don't need a systemic liver/lipid-hitting drug for this.
The foundation is hygiene, this does more than people give it credit for, especially for back/body acne on cycle. Shower daily, and especially right after training sweat sitting on your skin with the androgenic load you're running is a recipe for back and shoulder acne, so don't let post-workout sweat dry on you. Change your towel weekly (a damp, reused towel is a bacteria reservoir you're wiping across your skin). Change your sheets and pillowcases weekly too you sweat into them nightly and then lie in it, which feeds back/shoulder acne directly. Don't sit around in sweaty training clothes. For a guy on cycle with elevated sebum and androgens, this baseline hygiene genuinely reduces the bacterial and sebum load that drives the acne, and it's free with zero downside.
Topicals this is where you actually treat it without going systemic. Salicylic acid (acide salicylique) is your workhorse for body acne it's a BHA, oil-soluble so it penetrates and clears the pores, and you can get it in body washes for back and shoulder coverage. A salicylic acid body wash used daily in the shower hits the areas accutane was treating. Retinol cream for the affected areas helps with cell turnover and is the topical version of what accutane does systemically much gentler, no liver/lipid hit. For body acne, a stronger topical retinoid (adapalene/Differin, available OTC) is more effective than plain retinol and is the go-to for this. Benzoyl peroxide is also worth adding it's antibacterial, kills the acne-causing bacteria, and a benzoyl peroxide wash on the back/shoulders complements the salicylic acid well (just be aware it bleaches fabric, so white towels and sheets).
Now vitamin B5 (pantothenic acid) is great for acne. The B5-for-acne approach exists and some people swear by it, but the dose is the thing to get right: the protocols that show an effect use mega-doses we're talking grams, like 5-10 grams per day. The acne protocols use 5-10g (1000-2000x higher). So if you consider trying B5 for acne, it's a gram-level megadose, not milligrams. Honest caveat though: the evidence for high-dose B5 is mostly anecdotal, the megadoses can cause GI upset, and it's a lot of pantothenic acid so it's an optional experiment, not a cornerstone. I'd lean on the hygiene and topicals as the real plan and treat B5 as a maybe-add if the topicals aren't enough. But if you do it, grams not milligrams, or it's pointless.
Now the prolactin, we need to control it. So let me hit it hard: uncontrolled E2 plus rising prolactin on a 19-nor is exactly how you get gyno that appears overnight. Deca's progestogenic activity plus estrogen is the classic deca-gyno mechanism, and it can come on quickly once it starts. So getting ahead of it matters, especially since you're committed to deca for 18 weeks and you've got crashed SHBG meaning more free hormone around.
For prolactin management P5P (pyridoxal-5-phosphate, the active B6) does lower prolactin and is a reasonable first-line a higher dose (200mg+) is what's used for prolactin control. And L-tyrosine as a dopamine precursor supporting dopamine 3000 mg (which suppresses prolactin) has a logic to it, since dopamine is the brake on prolactin. Those are the gentler interventions. If prolactin climbs significantly or you get symptoms despite them, low-dose cabergoline (0.25mg) is the stronger tool just dose it conservatively, you don't want to crash prolactin to zero (it has roles in wellbeing, libido, immune function). Note caber cause more LVH (left ventricle hypertrophy).
But masteron is the better upstream move and I'd emphasize it: masteron has anti-progestogenic/anti-estrogenic activity at the receptor level, so it works against the deca's progestogenic gyno pathway at the source rather than chasing it downstream. Adding masteron to a deca cycle is a genuinely smart way to blunt the progestogenic sides while also helping with E2 (mast's anti-estrogenic effect). So for a guy on 18 weeks of deca should be worried about prolactin/progesterone gyno, masteron earns its place it addresses the mechanism upstream. The one tradeoff to flag: mast is a DHT derivative, so it can worsen hair loss for the genetically susceptible, and it'll add to the androgenic load (relevant for the acne too, ironically). But for the prolactin/gyno control on deca, it's the right upstream tool.
The critical point to hammer, don't let E2 run uncontrolled while prolactin climbs that combination is the overnight-gyno setup. So the play is to get your pending E2 reading (you're still waiting on it), keep E2 in a controlled range (not crashed, not high), and stay ahead of prolactin on the long deca run with P5P/tyrosine, masteron as the upstream blunt, and caber in reserve if it breaks through. Watch for the early gyno signs nipple sensitivity, itching, a lump forming and if they show, act fast, because deca/progesterone gyno doesn't give you a long warning. Getting the E2 number and adding masteron are the two highest-leverage moves here.

Always a pleasure to help out a brother out !