posted Wed, 09/16/2026 - 17:14
613
+ 6 Lipids under control
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Alright so here’s my last 3 bloods. July I was running a trt dose of 180mg weekly I was fucking around with superlean pwo few times a week which I approved to the higher ldl I ended up proceeding with my cycle and ran 400 test 200npp 200 mast 200 primo and was running mtren 3 times a week and superlean twice a week that really took my lipids to shit so I dropped them but upped my doses to 550 test 450 npp 300 mast 300 primo and added daily 10mg eze 10mg rosu and 20mg Cardarine and man I couldn’t be any happier with my results not only do I feel amazing my lipids are back into reasonable ranges. I’ll definitely be implementing Cardarine in all my blasts going forward and see how just that does for my lipids but man what a good feeling.
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This is great stuff your doing. Thanks for the time and service you provide to us brother. Great work
Glad to see your protocol brought numbers down to pretty reasonable levels. Especially while still on. Dr just put me on Pitava, only 1mg to start. Curious to see what that does. Numbers weren't terrible, but with family history getting on top of it
Thank you for posting bloods. great help to the community
I’m just envious of that hematocrit! Mine does not want to cooperate
I’ve always been lucky there that’s why my next run will be some bold cyp
Cardarine scares me, your not worried about it?
Not at all the dosages that they were giving the mice that caused cancer was 10mg/kg daily so a 200lb person would have to take about 900mg daily I will never get anywhere close to that. 20mg has done exactly what I needed it to do
That math is completely off because you cannot directly multiply animal milligrams per kilogram by human body weight. Pharmacology uses allometric scaling based on metabolic rate and body surface area to calculate the Human Equivalent Dose. Using standard FDA conversion ratios, 10mg/kg in mice translates to roughly 73mg for a 200 lb human, and the actual GSK trials showed systemic malignancies in rats starting at just 3mg/kg, which is only about 43mg in humans. Most importantly, clinical pharmacokinetic data proved that circulating blood exposure at a standard 20mg human dose is only two to three times lower than the plasma levels that caused widespread multi organ tumors in the rodent trials.
I did not write all that above, I am not that much of a egg head to be able to come up with all that on my own lol. I asked AI about this topic about the ratios between rodents and humans based on the trials done, and why the drug was discontinued. You're my bro, so I just don't wanna see anything bad happen is all. I'm just trying to look out for yah.
There’s also this
“ The often-cited 2010 mouse gastric-cancer experiment deliberately gave mice DMBA, a chemical carcinogen, before GW501516. In that experiment, GW501516 + DMBA produced rapid gastric tumors, while GW501516 alone did not produce tumors during that relatively short experiment. So that particular study primarily demonstrates tumor promotion after carcinogen exposure, not that Cardarine independently initiated cancer. ”
Yeah but that was a completely different trial that was separate from the GSK trial. The GSK trail was done with multiple rodent groups using different dosing of Cardarine without DMBA, they still developed spontaneous multi organ malignancies across all dose groups.
The lowest dosage was 3mg/kg in female rats which is the equivalent to 44mg a day but there aren’t any human studies so all of this can be completely useless as our metabolism and digestive systems are a lot more complex and there wasn’t any studies done on what the standard human dose schedule is. But I can see your concerns
If there is even a low chance of cancer I wouldn’t take it. I just don’t think the risk is worth the gains you could get from it. You can take Reta, Clen, T3 and many other compounds that aid in losing weight that are all proven to be highly effective, and no possibility known of them being cancerous. Now I guess the Reta at the current moment is still questionable.
Do you take HGH? And you consider the “possibility” of cancer from cardarine more dangerous than the known myocardial damage from clen?
The difference between GH and Cardarine is one directly creates it and the other fuels it. Two completely different things my guy.
I’d have to disagree. It’s yet again other scenario of one being known vs unknown. There is no clinical evidence with humans to make the assertion that they are somehow synergistic in cancer causing pathways.
And also, not two different things if cancer is the outcome in both scenarios. Whether it’s fueled or created is inconsequential once you have cancer.
What I’m trying to get at is why is cardarine the enemy when it has no human clinical evidence supporting your stance? Nor does it even have anecdotal evidence supporting it yet. But the other two things you proposed do, in fact, have the data to support negative outcomes.
We always use rodents in all studies as a first pass at seeing if we will take a pharmaceutical drug to the next step. This is just standard practice. Regardless of real human studies or not it’s a highly toxic compound that causes cancer and the likelihood of a cross over within a human test subject would result in the same outcome.
You said it’s inconsequential if the end result of each scenario is cancer, I disagree. It makes a huge difference between the created cancer within your self using a compound that is already known to give cancer directly, and one that is accelerating your cancer growth that you got from other means entirely with the use of GH. Which btw your body already creates endogenously so it’s gonna grow regardless. On one hand with the Cardarine you win the Darwin Award, congrats your stupid. On the other hand it’s just sad and “oh fuck bro that’s horrible”. Because the growth of cancer in the GH scenario was there all along anyway.
These are two very different scenarios, one is completely avoided the other one was just a time bomb.
The other things I mentioned like clen, t3 do have reported factual negative outcomes, because they have been highly studied and have been green lit for humans. At least when we sign up to use these compounds we know all the negative and positive effects. I know the risks I take and how to mitigate the possible negatives because of how much research has been done on them.
This is like SARMS, would you take a random SARM that has had no studies done on it, maybe just a few with rodents? Or would you take what we all know to work well, and give’s results? Granted it has negative sides as well nothing is side effect free. This would be for examples sake (NPP)?
One is a mystery that turns into a complete gamble, the other one is a complete calculated risk?
You can’t use other medications allowed for human use as your argument when we use them at doses 10x higher than what’s studied. It turns those scenarios into mysteries as well and no longer calculated risks. Taking a replacement dose of GH = calculated risk
Taking 8 IU GH = mystery
Same can be said for test, NPP, Drostanolone, or any other AAS developed for muscle wasting, aids, burn victims, breast cancer, etc.
Except maybe anadrol. They were slamming people full of that one lmao. We take less in our world than what they were giving clinical patients.
I used these other compounds as argument, because you you created a parallel between those said compounds and Cardarine. Pretty much saying it seems to be worse to use those compounds cause there is already factually known negative sides with them and yet Cardarine has no substantial evidence within humans for use to smear it like I have. It's funny cause now it just seems like you are back peddling a bit.
Taking 8 IU GH = mystery?? Idk about that people have been doing that for 40 years now, the amount of anecdotal evidence on what it does at this dose is insurmountable.
I guess if you wanna pop Cardarine (I hope not) then go ahead its on every source page practically. I was only on here advocating against it cause someone who I see as a friend is using it, and I just wanted to make sure he understands the ramifications of the compound. He probably already does he's a grown adult, so I'm probably wasting my own time lol.
I’m not back peddling on anything. End of the day any negative side effects of cardarine are purely conjecture. Anecdotal experience collected in a non clinical setting, which controls other variables, even if it’s “insurmountable”, can only lead to correlation and not causation. Without knowing for sure what cardarine does in humans, then your argument should mean you do not and have not ever taken any current peptides on the market either. There is equal fear mongering on bpc157 and tb500 causing cancer cell proliferation.
I’m merely pointing out the fallacy in saying don’t run cardarine (at human dose equivalents lower than rodent cancer inducing doses) but instead run clen. A notoriously heart dangerous/damaging drug when abused. And it’s barely prescribed across the world compared to salbutamol. The only reason clen is prevalent still is due to the abuse of it from our community. Not for treating asthmatics.
Also didn't they give them the super high doses for something like the equivalent of 2 human years straight?
No the trial took place for a total of two years before they trashed the compound due to every dose from low to high was cancerous.
I just looked into those GSK studies in more detail using AI. I totally understand why you would not want to take it, and I agree that the cancer concern is legitimate. I just think that description goes further than the published information supports.
The studies lasted 104 weeks, but only short conference abstracts were released, not the complete studies. The abstracts say there were treatment-related tumor findings at every tested dose. That does not mean every animal developed cancer. We do not know what percentage of the animals developed tumors or when during those two years they appeared.
For a 200-pound human, the lowest female-rat dose works out to a rough human equivalent of about 44 mg per day, while the lowest male-rat and mouse doses work out to about 74 mg per day. They received those doses every day for essentially their entire adult lives. As a rough comparison, that would be more like a human taking Cardarine continuously for decades, from early adulthood into their 60s or 70s. That is quite different from someone using 10 mg per day for several weeks.
I am not saying that makes Cardarine safe. We simply do not have enough information to quantify the risk of short-term, lower-dose human use. I completely understand deciding that the unknown cancer risk alone makes it not worth taking, though.
Yeah we just don’t know when it comes to humans, and I don’t wanna find out either. I enjoy life, and bodybuilding too much to let cancer take me out over something I could have completely avoided.
But the lowest dose used was equivalent to 44mg so if it’s been found to be effective at 20mg in humans why not see what 1.5mg/kg does for rats
I’m guess they didn’t go lower cause it wasn’t worth the extra research, time, and money invested to see. It’s a very hard sell for anyone when you tell them it’s cancerous, regardless of dose or not.
But yet it’s been proven that ultra processed foods cause cancer and people eat them everyday lol
You guys are going to make me a hypochondriac, jesus.
Welcome to my world lol
Hang out out at the laundry mat?
Im really tired, I dont get the joke
Slim shady lyrics lol
True can’t argue that lol, same with cigarettes and alcohol.
Saw you @ 999. I was your 1k
remember that forever lol
Thank you Oscar +1 my friend lol