Okay so I have been checking up on how estrogen and prolactin effect each other. Like keeping estrogen under control keeps prolactin under control (from what I understand). I know some of you will freak out about this but I have never used an ai or caber or prami on any cycle and (knock on wood) never had any issues. I guess I'm not too prone to estrogen or prolactin sides but I know some are.

I prefer to keep stuff simple and not take unnecessary meds (unless needed). Imho people take too many meds on cycle when they are not needed. I'm not saying don't be prepared or don't have stuff on hand just in case. Especially if you already know your sensitive or prone to getting them. I'm just sayin you don't need to ALWAYS run an ai or prami on cycle.

Some of the information I posted below definitely goes against the norm and I thought it was very interesting. I'm not saying I believe or confirm the posts, I just feel its worth talking about.

This was from UK Muscle and the person who posted was Ausbuilt. Below that is a quote from Bill Roberts (who some say is a aas legend and genius and others say he is a moron). Again I'm just interested in peoples views and opinions.

Deca dick is not caused by high prolactin:

http://www.ncbi.nlm.nih.gov/pubmed/18082071

see its not prolactin rise that causes ED. it may play a part, but its not a direct cause. Further, when it comes to deca dick, its due to DHN:

"...Erectile dysfunction is attributed to the weaker action of dihydronandrolone in the penis since dihydrotestosterone is a known sexual modulator."

from: http://en.wikipedia.org/wiki/Nandrolone

Basically DHT is a strong androgen, and causes erections (think proviron) and when you take test, 5-7% of all test converts to DHT (and gives you erections) where as 19-nortest derivatives such as deca and tren reduce to DHN which does the opposite...

At any rate the whole think with tren/deca and prolactin is... a BIG misunderstanding...

There is some pretty basic misunderstanding out there, with people repeating "bro science" and saying they have "prolactin" gyno from deca or tren, which is physically impossible:

Prolactin "...stimulates the mammary glands to produce milk (lactation): Increased serum concentrations of prolactin during pregnancy cause enlargement of the mammary glands of the breasts and prepare for the production of milk. However, the high levels of progesterone during pregnancy suppress the production of milk. Milk production normally starts when the levels of progesterone fall by the end of pregnancy and a suckling stimulus is present

thats from: http://en.wikipedia.org/wiki/Prolactin

You should note that, HIGH progesterone INHIBITS prolactin!!

Then, 19-nortestosterones , are classed as progestins:

"Clinical studies have shown" Deca Durabolin..." (a 19-nortestosterone derivative) "... to be effective in treating anaemia, osteoporosis and some forms of neoplasia including breast cancer, and also acts as a progestin-based contraceptive... Estrogenic effects resulting from reaction with aromatase are also mitigated as a result of the drug being a progestin."

from: http://en.wikipedia.org/wiki/Nandrolone

And if you read this, 19-nortest has MINOR activity in aromatisation in men:
http://www.sciencedirect.com/science...39128X66910129

Now what DOES cause breast development?

"While estrogens are present in both men and women, they are usually present at significantly higher levels in women of reproductive age. They promote the development of female secondary sexual characteristics, such as breasts, and are also involved in the thickening of the endometrium and other aspects of regulating the menstrual cycle. In males, estrogen regulates certain functions of the reproductive system important to the maturation of sperm[10][11][12] and may be necessary for a healthy libido.[13][14]"
from: http://en.wikipedia.org/wiki/Estrogen

Now progesterone:
"...Progesterone has a number of physiological effects that are amplified in the presence of estrogen. Estrogen through estrogen receptors upregulates the expression of progesterone receptors.[26] ."

also:

"Progesterone is sometimes called the "hormone of pregnancy",[35] and it has many roles relating to the development of the fetus...In addition progesterone inhibits lactation during pregnancy. The fall in progesterone levels following delivery is one of the triggers for milk production." (folks please not, as noted above in prolactin- high progesterone INHIBITS prolactin, low progesterone lets prolactin rise).

both the above from: http://en.wikipedia.org/wiki/Progesterone

So to summarise that:
1. 19-nortest derivatives (deca & tren) are progestins
2. High levels of progesterone INHIBIT prolactin
3. Progesterone only exerts the majority of its effects in a high oestrogen environment.

So, where the F**K do people get of saying take CABER to suppress prolactin when taking 19-nor steroids??? Taking a 19-nor steriod AUTOMATICALLY INHIBITS PROLACTIN!!!

That was from UK Muscle and this is from Bill Roberts:

Those claiming "prolactin" apparently never do so from actually measuring prolactin. 

There is no evidence that trenbolone increases prolactin, and what evidence there is -- from the veterinary literature -- shows that it does not.