+ 6 JUMPSTART
Ok first let me say, Mods I have put this in the general for one reason as opposed to putting it in pct. The pct section is primarily going to be for newbs and I consider this next level debate for the advanced that might eventually impact the newbs if protocols evolve or change as it pertains to pct.
I was about to give a pct protocol in the cycle section when I noticed a new member who has some pretty good general information had once again posted 10 consecutive days use of hcg in the post cycle, of which I don't agree. So rather than reply given it is becoming a habit I thought a good debate here would be better. First let me say, yes, I disagree with post cycle use of hcg and I will lay out those reasons for debate but I'm not trying to say zewi is wrong and I'm right. I am an evolution type person and will follow the science and in fact try new things almost every cycle. So having said so, zewi belives in his pct and may have had great luck with it and I may have had great luck with mine before I went trt but that never means we could not have had better luck if we had discussed ours together. Good science usually incorporates good ideas and slowly morphs to better idea's.
So here's the problem and I would like everyones thoughts on this, hcg will slow the production of LH from the pituitary. Now let's talk about why that is important or even debate if its true. First of all what occurs when you begin popping those test shots? Without going into an Anthony Roberts science class, there is simply a negative feedback loop that occurs and tells the test side of the axis, "we don't need you right now". This of course means the message got to the pituitary and there was a change in hormone at the LH and FSH level. Alright, skip tp to last pin in a cycle, the gear is gone and we need to get the message to the brain that test production from the free train is over, time to get that ass back to work. So now we hit the all important "dead zone" right? No test coming in the body via the pin and the natty is offline and went in such a deep sleep even with the positive feedback loop coming into play, its tired, its pissed, it simply does not want to get out of bed!!! Lazy bastard!
So what to do? Well first up we recognize the biggest problem. Again, without going to deep in the mind numbing science the root cause is the pituitary is not generating the LH and FSH response needd to prompt the gonads back to service. There's a complicted step before that but it serves no purpose to the discussion here so let's get to it. We have to get the pituitary to function here and do its job. Now many think as I believe zewi does, and may be right btw, pump those balls up and it all happens. Well not so fast for me, yes there is benefit from hcg to the nuts but nothing to prove it has any impact on the axis and natty reboot. In fact, hcg will increase estrogen and at the same time hinder LH and LH and FSH have to come back into play for the positive feedback loop to even begin to prompt natty. So you ask, "well then why use hcg at all"? That's a good question and yes, there are plenty of good reasons to use hcg. The sex drive, the health of testes in atrophy and many more. So I think nolva is your best friend in the post given it plays many rolls which include helping the estro block , sending message to the pituitary estro is not wanted and sending message to the pituitary to bring LH and FSH back online. Clomid is not a bad idea either given the double whammy but not nearly as effective at those things as nolva, and in fact maybe no effect at all in terms of pituitary stimulation that nolva has been proven to have. Another fact is that aromasin might play nice roll in the post but that's another time.
So here's the debate I have with zewi. My thoughts are this, use the hcg either whole run or in the final two weeks to keep or bring back the nuts. Incorporate an AI (aromasin always for me) to prevent the known spike effect hcg has on estro/shbg and then drop it opting for one of the known effective pct plans ALWAYS incorporating nolva as it will actually be the ultimate magic tool in stimulation at so many levels, again, I understand the debate around LH as it pertains to hcg but I simply do not understand why would want to bring LH online and at the same time incorporate a drug know to prevent that to some degree right at the time we prepare to crash. Now let me say this, if we were to run the 10 day protocol following a long ester test it would be a debate I'm much more inclined to lean towards but following a short ester test I just don't see it. Bring the debate and bring any other ideas you have be it aromasin in the post etc, just have some scientific discussion with your opinion so we can try to follow it.
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56toomark
sgtstedankoI fell asleep halfway through this, so like usual, I have no clue what we're debating
I've never used hcg. Everything I've read agrees w/you tread. It looks like It's best to kind of bounce it in & out throughout cycle up until Pct. My nuts are too big anyway, I have trouble finding trousers :(
tread-mI've said it before and I'll say it again, that sarge has some big ass balls!
sgtstedanko: ) spread the word, brother. Maybe Lauren will take notice
shweaty bawlls
sgtstedankoLol... Nuts
zewiI like this Discussion tread-m. True something’s work for others and something’s not, and I’m all about changing my PCT if it benefits me and others. I’m not here to say my way is better or yours either. My goal is to come up with the best.
The reason I like HCG (after cycle) is it keeps my balls in good shape, but I also keep taking Arimidex during this time after a Cycle so estrogen won't spike . Also I understand the effects that HCG has on the Pituitary gland and that estrogen tells the pituitary to stop making more LH.
However, I add Clomid to the mixes which blocks estrogen at the pituitary, so In turn makes more LH—(nuts make more test). The male HCG level should be no higher than 5 (normal is 3), if it isn’t it will take longer for Clomid and Nolvedex to do its job. However, doing a little blast at the end of the cycle makes sure that my HCG level is where it should be when I start my Clomid and Nolvadex—So my nuts are ready to get started right away cause they are in good shape and my HCG is in the correct level to start producing LH---my normal test.
Can’t wait to see where this goes. I’m looking for much input from yourself Tread-m, and others.
tread-mGood good, love the discussion here. Alright first up estrogen did not really stop the LH production from the pituitary so I think there is flaw in that thinking. What stopped LH was the shutdown brought on by negative feedback loop with the introduction of artificial use of test. So to say by using clomid (which is not an atii e at the piruitary btw nolva is however even though both serms very different) at the pituitary will somehow jumstart LH is incorrect IMO. The jumpstart will occur when there is no negative feedback loop and when suppression of LH is not present. So as you use a short ester as the person was in the thread that prompted our discussion here, you have dropped free test plasma levels dead in 48 - 72 hrs. After last injection. Now we have this hcg coming in which is suppressing LH so we are preventing the LH upstart an additional 10 days so as to artificially cause the actual axis balance natty to postpone an additional 10 days,, make sense? By using the clomid you are simply blocking receptors. Yes, the nolva is stimulating LH production but its like saying I'll jumpstart my car but won't hook up the cables to the good battery at the other side. Does not work in my opinon, counterproductive at best. So as to it keeping your balls pumped up, how does that benefit you and more importantly, how does benefit jumpstart? I agree with the use of hcg in the cycle for healthy balls, fine, but understand with artificial use of test LH is off anyway. It does not matter in the cycle, LH is off anyway and we have the artificial means of test making it not matter. In the post following a short ester it very much matters given we now no longer have the artficial test, LH is off and we need it back and hcg is not helping that happen. Its actually hindering it from all I know. I agree with good healthy nuts but that can be achieved either on cycle or in the close of the cycle without worrying about the extra stress on LH in the post. Now if hcg was a prompt for LH then I'm all in, just seems backwards to me here though. My goal btw is not to call you out , in fact Anthony Roberts touts an even stronger hcg post than you and I don't buy into it either. The reason for that as much as I love his work is I'm not a parrot for anyone and simply don't think he has this one right as much as he is right IMO on so many things. So you are in pretty good company I guess but this LH is unresolved for me, there is simply not a single reason I can find scientifically to support it. He himslf agre's that LH is suppressed and then goes on somewhere to justify the additional stree. Simple for me, I want LH back to normal becausee we know its what will bring natty back online and hcg hinders that. Again, I am not opposed for any other reason than fact and this is not who"s right or wrong, its what is right? I want to understand it , I really do , its just not there in fact at this point in discussion.
zewi“you have dropped free test plasma levels dead in 48 - 72 hrs” That’s correct however, if you have been taking HCG during cycle this isn’t really the case is it .
Sure we are talking about the pituitary and the effects HCG has on it. However, you are also forgetting the effects it has on stimulating the leydig cells of the testes. So hcg stimulates the testes in the same manner as LH. Hcg uses the body’s own Bio stimulating mechanisms to increase plasma level during the use of hcg. The HPTA (hypothalamic-pituitary-testicular axis) regulates the level of testosterone in the bloodstream, and, the hypothalamus produces gonadotropin-releasing hormone (GnRH), which stimulates the pituitary gland to release Leutenizing hormone (LH).
HCG effect on the pituitary is not the HCG as it is the estrogen—HCG has been known to increase estrogen when people are on cycle. Thanx Arimidex --- arimidex (anastrozole) lowers estrogen levels in men, slowing the growth of breast cancer and increasing testosterone levels in men. One of the hormones produced by the adrenal gland is converted to a form of estrogen by an enzyme called aromatase. Arimidex suppresses this enzyme and thereby reduces the level of estrogen circulating in the body.
Armidex is the most important thing to have when doing HCG, and a cycle. I believe it should be used every cycle. And again why I say you continue using Armidex when using HCG in PCT.
tread-mOk its off topic but let me add for you, aromasin much better choice. You will over time build tolerance to arimidex and need more and its less effective. With aromasin you not only don't build tolerance ,you actually need less over time and an added benefit ,it actually decreases gyno prone predisposition in a way adex won't to a great degree. Ok, you have bought into the myth I'm afraid. You have some Anthony Roberts mixed in there and I don't mind parroting but let me explain this as will he or anyone else. Hcg on cycle, off cycle , alive ,dead, coma, whatever the caswe....is not keeping you axis from getting shut down ever. Its a myth and proven over and over. You can run as much hcg as you like on cycle and post cycle and you go 10 day pst cycle after running prop your shutdown brother, period. That's a,ctually where this whole hcg in the post argument began, people thinking it stops the full shutdolwn or decreases it etc, until of course,all the bloods came rolling in and woops...nope didn't work. So I do appreciate the thoughts here but still no science as to the hcg in the post. The problem is you have no test to increase here in the early post. There simply is none. What you are referring to in adex is if someone were not on cycle at all and ran adex, it does via mechanic essentially increase test but its simply by lower the competitionin etrogen and sex hormone binding globulan which in turn allow a greater number of test ikn ratio. Than binders to hit receptors. Same with hcg in an off cycle mode where natty is normal. The point I thi nk you are missing is you are shutdown, period. If hcg prevented shutdown we would not even have pct. You would run hcg on cycle ,boom , you're still at natty levels and off to the races. The simple question for me is this again, we have LH suppressed which is what will ultimately bring back natty via prompt and feedback loop so why would we use hcg which will hinder that in the post when running short ester in particular. I think you may be confused in thinking estrogen has anything at all to do with it. We want estro low in the post to keep SHBG low so we are not robbing natty test as it reboots and of course to keep the muscle via no receptor competition from estro as well as gyno concern, it still has little to do with reboot itself. Seems like you are confusing its relationship to reboot, of which there is none. Healthy debate hre brother, don't take me wrong ok. I like you are reading about this I just think you are confusing the relationship chain in the hormones a bit .
zewihttp://www.eroids.com/forum/steroids-qa/pct-anti-estrogens/hcg-in-all-it...
has a much better write up than i can do..
tread-mOk I'll take this one first then the other one in a bit when I get a little more time to go in depth (at work). This one is pretty random more a description of hcg although does address the LH suppression I refer to and keep in mind, the post cycle here refers likely to a long ester in this one when the test is still very active on the 2 week protocol post cycle...not even really post cycle. Your advice was on a short ester cycle which would be the equivalent of using it 3 and 4 weeks off cycle. ....so still, this does more to confirm my thoughts as far as I can tell. This other one I know I've read but I'll get a few here and go back over it and we can discuss it. This is the sort of thing I wanted here, hopefully more chime in eventually.
zewiyou win.. I have nothing to add... I will stop responding to PCT questions.
tread-mLol...nooooo, don't do that brother. This is where its at seriously. Everything ever put to protocol had serious debate involved. Listen Roberts touts this thing so I'm seriously wanting to understand it so don't take my arguments as "I'm right". I just think I'm right until I see the light and everything I do know is a result of that and subject to change. This LH is just hard for m to get past, not that I can't though if something shows up. I would love to see LH and test levels at the exact end of this protocol and then again without hcg in thise 10 days...maybe there's something there to surprise me. Still hope we get more debate on this and I don't ever want to shut you down brother...debate is all we have! Lol
zewiAlso here is the link to William Llewellyn PCT
http://purepeptide.com/#/pct/4549235991
tread-mWhat happened here, link changed hell I know we can buy the shit! Lolol