Don't trust those Rythm numbers. I ran the test three times and got some inconsistencies; the last time, I made sure to get the sample taken and go to a lab to run the same tests. There were bizarre discrepancies—like ferritin at 600 at the lab versus over 1400 on the Rythm test, and HDL at 109 versus 146 on Rythm. The numbers were completely off.

Good to see the follow-up brother, and the trend is genuinely moving the right direction. Let me give you the honest read on where you are versus where you were, because the progress is real but the picture's a little more nuanced than "lipids improved."
What's clearly improved and this is the headline:
Your ApoB went from 156 at peak to 115. That's the number that actually matters for cardiovascular risk, and a drop of that size is meaningful, real progress. ApoB is the marker that tracks the atherogenic particle count, so moving it from 156 (high-risk territory) toward 115 is the single most important improvement on this panel. The supplement stack you built fish oil, citrus bergamot, psyllium husk especially plus presumably dropping any orals, is working. The psyllium husk in particular is pulling weight here; soluble fiber binding bile acids and forcing your liver to pull cholesterol is a real mechanism and your ApoB drop reflects it.
LDL down from 181 to 162, HDL recovering from 25 to 36.8 both moving the right way. HDL climbing back toward your baseline of 41 is good, and for a guy with your genetics on 600 test, 36.8 is a reasonable place to be mid-cycle.
BP in the 120s/70s this is a real win and it's the thing we talked about way back. You fixed the BP. That's done, and it's probably the single biggest reduction in your actual near-term risk. Well done on that one.
Now the two things that need flagging, because they're moving the wrong way:
Your E2 climbed from 64.7 to 91. That's a notable rise and it's now genuinely elevated. The question is why did you drop an AI, change your test dose, gain body fat (more aromatization), or something else? E2 at 91 on a standard assay is high enough to potentially cause symptoms (water retention, nipple sensitivity, mood, BP contribution) and it's worth understanding what drove the jump. It's not an emergency, but it's the thing on this panel that changed most in the wrong direction, and going from 64 to 91 isn't random something shifted. If you're asymptomatic, you can watch it, but I'd want to know the cause. If you've got high-E2 symptoms, that's the lever to look at. Worth noting the E2 rise could also be partially driving the next issue.
Your hematocrit crept to >51% and it's been steadily climbing — 49.7 → 50.45 → >51. This is the trend I'd actually watch most closely now, because it's the one consistently moving up across all three panels. You're on 600 test, your hematocrit baseline was already on the higher side at 49.7, and it's now over 51 and climbing. That's the trajectory that, left unchecked, gets you into donation-required territory. It's not dangerous yet, but it's the clearest upward trend on your panel and the one most likely to become a problem on a 600 test cycle. Keep an eye on it, stay hydrated, keep the cardio up, and start thinking about a blood donation if it keeps climbing past 52-53%. Don't let it sneak up the way it did for some of the other guys.
The finasteride note:
I see finasteride in your stack now wasn't there or wasn't mentioned before. Fine for the hair, just be aware it's not affecting your lipids or the things we're tracking here. No issue, just noting it's new.
The genetic dyslipidemia reality check, since this is the through-line for you:
Here's the honest long-view, and it's the same conversation as before. Your ApoB at 115 is much better than 156, but for someone with your genetic baseline who plans to keep running gear, 115 is still above where you'd ideally want it (under 90, arguably under 80 for someone actively managing cardiovascular risk). You've proven you can move it significantly with the supplement stack and lifestyle that's genuinely good and it shows the interventions work for you. But you're also a guy whose LDL was 137 with HDL 41 before you ever touched a cycle, which means your genetics are working against you, and the supplements are doing real work but may not be enough on their own to get you to truly optimal numbers long-term.
This is the same place we landed before: for someone with your genetic dyslipidemia who intends to keep cycling, the conversation worth having with a doctor is whether a low-dose statin (rosuvastatin or pitavastatin) or ezetimibe added to what you're already doing would get your ApoB down into genuinely protective range. You've shown the lifestyle and supplement approach works it took you from 156 to 115 but the genetics mean you might need the pharmaceutical assist to close the rest of the gap. That's not a failure of your protocol; it's just what your genetics call for. The supplements got you most of the way; a statin might get you the rest.
Bottom line:
Real progress, and you earned it ApoB down 41 points, HDL recovering, BP fixed. The stack is working. Two things to watch: the E2 jump to 91 (figure out what drove it), and the hematocrit creeping over 51 and climbing (this is the trend to monitor, donate if it keeps rising). And the standing genetic-dyslipidemia conversation: you've proven the interventions work, but your genetics may need a low-dose statin to close the gap to truly protective ApoB numbers worth raising with a doctor. You're doing well, the trend is your friend, just keep the hematocrit honest and sort out the E2.