posted Fri, 09/12/2025 - 02:56
996
+ 4 Bloods on test e & hgh
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Currently using weekly
metapharma test e 600mg
para pharma masteron e 400mg
Para pharma deca 300mg
Letro half tab 1.25mg
Puca brand HGH 12.5 iu split 6am & 9am (IM shot)
Tirz 7.5mg wkly
Meta test is working great shame they closed up.
Im confused about the hgh igf number coming back in range with the volume of hgh im using im suprised, I dont what to make of it as coa on this brand was 35ius when kits sold as 24ius.
I did want to run gh serum but the company i used to buy didnt offer it.
Bloods was drawn 10am.
Ordered from:
- Bookmark
- 4
- 0



Your testosterone levels are really good for 600 mhs a week. Mine are between 2,400 depending on how long after my shot I take it. 2,400 is when it’s been 4 days after injection
I did run bloods day after my shot bud so i can see how you got 2400s 4 days later is still solid.
I would assume that the Letro you are taking is lowering your IGF-1 as it can definitely do so. Are you taking anything else like metformin or berberine?
No bud nothing else but with my estro being in higher range id expect some igf bump surely.
Im defo going re test on diff brand and see again if anything changes.
Strange enough iv had zero sides running that high dosage i thought maybe as im doing IM AM shots its causing little to no sides inc clean diet.
I remember back few years ago 5 to 8iu id have to give up as would get alot tightness and joint aches doing subq AM.
Urgent, Urgent - You are about to turn into a Flaming Ball of Testosterone!!!
And Still nobody knows why a great Source did a runner .....
Well spotted, brother! Meta was always strong … every vial I saw was either properly dosed or even slightly over. Absolute quality across the board, and their customer service was actually solid too. That’s why it never made sense for them to just disappear and screw everyone over like that.
To me, it doesn’t look like something planned or strategic. More like something personal happened behind the scenes … a fallout, a panic move, maybe even some internal drama. Could be wrong, of course, but no one runs a source that solid for years and then suddenly pulls the plug if it’s all about business. It just doesn’t add up.
Appreciate the response guys im baffled as IM peak time i was told is 60 to 90 mins hence my protocol.
I have some baboon pharma 10ius kits im try that 10ius split and re check igf & gh serum this time in a month then will see if its me or the hgh.
Every bloods iv ever done has elevated igf when iv used less 5 & below ius so i was alittle shocked on this result tbh.
My liver values are not elevated much as almost normal range and i let my estro run up high too as i always keep that around 20s.
For IGF-1 timing doesnt really matter for blood tests. GH serum is where that matters but there mostly a useless test that I wouldn’t bother with. iGF-1 Levels peak and stabilize after a week on the same dose and you just test anytime after that really. I usually take my morning dose and test sometime that day.
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IGF-1 levels don’t peak 60-90 mins after a IM injection. The first thing that’s going to peak is GH serum levels which is around the 3.5hr mark. For IGF-1 to peak GH serum has to have already peaked. So IGF-1 peak will be at best around 6-12hrs after an IM injections maybe longer.
Hey Buddy - I just wanted to bend your ear for a second if I can. In regards to us talking about BP medications before and the fact that Telmisartan inhibits the Angiotensin 2 receptor (the one that causes/facilitates collagen formation in the heart), if you were to take an ACE inhibitor like Ramipril would that also do the same thing as in blocking the no.2 receptor since it blocks the angiotensin converting enzyme in the first place so nothing can in theory lock onto that receptor anyway?
Like would it have the same effect on potentially preventing collegan formation as ARBs do by blocking the angiotensin II type 1 receptor, but instead by preventing the conversion of angiotensin I to angiotensin II? I think so. If collegan is produced after activating the AT1 receptor, which only happens If theres Agt II, then by blocking the conversion to Agt 2 with an ACE inhibitor should result in none being available to bind and activate the AT1 receptor.
However, and I don’t really know how this plays in here but the issue with ACE inhibitors and preventing any conversion to Agt II is the activation of the AT2 receptor has many cardio protective effects and in normal circumstances it’s counteracts the negative effects of the activation of the AT1 receptor. Whereas ARBs only block the bad AT1 receptor and still allow the good stuff to happen from the AT2 receptor. So like would we potentially be preventing collegan formation, but at the same time blocking the vasodilation effects of the AT2 receptor which reduces BP and inflammation, it also increases renal sodium excretion, cell proliferation etc etc. and idk the answer to that one without doing more digging. The answer is probably similar to why ACE inhibitors arent as popular as they once were
Yes thats what I was meaning that you have stated in the first paragraph, although I didn't term it that well lol Ahhh so in effect you could be eliminating one problem, but by doing that creating another one that didn't exist before Lol I ask as I have gone back to Ramipril again as I seem to get less side effects from it - I was starting to get an upset stomach all the time on ARB's and not feeling too great. On ramipril I barely feel any different at all, I was getting worried I was getting the ACE cough too in the past so stopped but I think it was probably something else. When I looked into that more it turned out the main people that got the cough were women and smokers. Its my understanding that the cough is the main reason why people stopped using ACE's and went to ARB's.
My bad yeah you correct gh serum peaks then igf.
With this igf-1 the hgh is bunk, i know that chase irons and others have normal igf-1 with 10+ hgh bla bla bla, but with this level of igf-1 there are 2 possibility: your hgh is bunk, you have liver cirrhosis. 99.9% it's that hgh is bunk.
@irongame427 what do you think?
More than likely, yes it’s bunk, or mostly bunk. However, dosing 6.25ius at 6am and then the other half just 3hrs later is not long enough between to get maximum IGF-1 response from the 2nd dose. Too much of the first dose is still active. Need atleast 6hrs between doses. If it was me and I was bulking I’d be doing half Am and the other half right before bed. But still that wouldn’t explain sub 200 IGF-1 levels on 12ius of GH.
I know Chase Irons IGF-1 levels were not super high on 18ius a day, but they were still fairly elevated right? Like 300-500? Still not as high as you’d expect on 18ius.
18ius daily! I’d need to rob a bank to pay for that course. I’m having 6ius and fat melts off fast. What’s the need to go above 6-8ius? Unless it’s seriously under dosed or you’re trying to grow your organs, fast track yourself to an early grave through diabetes etc.
I believe once you go 8iu's and above you can start adding some good mass with it similar to steroids - especially if you throw in the naughty stuff with it.
I’m sure you can but wonder at what point the results versus health risks diminish? Had great results at 6iu putting on size and melting fat without running you know what with it. Can’t remember but I may have gone up to 8iu last time and was very happy.
Once you start getting above about 6ish IUs you may have to start supplementing with thyroid hormones as GH in high doses affects that, your blood sugar may run high and you’ll have to take you know what to control that etc. the threshold is different for everyone though. Track those things via blood tests to be sure.
I always get almost 100 per 1iu back in the days.
But iv been running this protocol for over 10days so id expect more.
Im thinking it was a typo with 6am and 9pm.
That would make more sense. But wild would make this IGF-1 result even worse.
if I remember correctly not above 200, which is very strange because roughly each unit of hgh increases the IGF 1 by 100 until reaching a plateau at around 7 IU ( without insulin i'm talking )
There’s a guy who was at 1330 on 10ius a day! On 4ius a day he was still 580. Both taken in 1 shot before bed. Most insane IGF-1 response I’ve ever seen. But ya generally it’s 100ng/ml per 1iu and it does seem to max out around 700ng/ml. But I do believe not all of GHs anabolic effects are due to IGF-1.
But we may disprove that theory soon. I’m running some GH that another guy was under 200ng/ml on 6ius a day. So I said that shit must be way underdosed or partially biologically inactive. And he’s now running pharm grade GH. If My IGF-1 levels come back over 400 on this stuff on 4ius a day, as it Always is on 4ius of pharm grade GH, and his come back under 200 or close to it on 4ius of pharm grade GH then we can say somewhat conclusively that IGF-1 response is a lot more variable than we originally thought.
Keep us updated bro, very interesting